Biosciences’ Cynthia McMurray was recently quoted in a Nature Outlook article on the gaps in our understanding of the biology that underlies Huntington’s disease. In the piece, McMurray, a senior scientist in Molecular Biophysics and Integrated Bioimaging (MBIB), discusses possible mechanisms that give rise to the abnormal expansion of the DNA sequence cytosine, adenine, and guanine (CAG), potential functions of the huntingtin protein in cells, and the theory that chains of glutamine (encoded by CAG) make huntingtin extra sticky.
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